3,535 research outputs found

    The Johari Wonderland: The Fusion of Classic Literature and Film to Enhance Key Group Counseling Concepts and Counselor Reflexivity

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    The Johari Window is a model of relational dynamics and key to understanding group counseling. Counselors-in-training must grasp the concept in order to flourish as group leaders in the field. Therefore, how might this concept be explained in a way that provides clarity and enhances students\u27 reflexive skills? This narrative analysis explored in what ways, if any, an originally designed Johari Window experiential activity can help students obtain understanding of the topic. A summarization of Carroll\u27s (1865/2014) Alice\u27s Adventures in Wonderland and Other Classic Works as well as movie clips from Tim Burton\u27s production of Disney\u27s Alice in Wonderland (Lebenzon et al., 2010) were utilized to describe the Johari Window. Three points of data were collected. A group of twelve students enrolled in an introductory group counseling course participated in a group interview and reflective writing based on the experiential activity. Then, participant checks were conducted regarding themes and feedback. Key findings included new perspectives on the Johari Window, enhanced personal insights, and a plot of identity development based on the story of Alice and the participants\u27 narrative

    Exploring notions of genre in 'academic literacies' and 'writing across the curriculum': approaches across countries and contexts

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    The SIGET IV panel on genre in Writing Across the Curriculum (WAC) and “academic literacies” (ACLITS) has set rolling a discussion of the similarities and differences in the two traditions, the former originating in the US in the early 1970s, the latter originating in England in the early 1990s. This paper maps out some elements of each in relation to the other and to genre, which we hope will set in motion further discussions and cross-fertilization

    Aggregated End-to-end Visibility and its Application on Rapid and Automatic Outage Triage in Monitoring Microservices

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    In a microservice architecture, a user request can go through a large number of servers owned by several different teams before a response is returned. The request can fail due to failure in any of the servers. Troubleshooting an outage that affects the end user experience in microservice architecture can involve multiple teams and can take a substantial amount of time. This disclosure describes techniques to rapidly locate the root cause entity of a customer-facing failure to node(s) deep within the infrastructure of the service. Per the techniques, end user product teams mark requests with metadata known as critical user interactions (CUI). The metadata is propagated along with the request. Performance metrics are gathered from servers that the requests go through. The performance metric is keyed by CUI, server node, and peer node for every adjacent pair of nodes. These piecemeal metrics keyed by CUI together offer end-to-end visibility for a set of requests grouped by the CUI of the end product, enabling the rapid and automatic triage of an outage to an interior server without requiring domain expertise on the product or the server

    Marketing Percolation

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    A percolation model is presented, with computer simulations for illustrations, to show how the sales of a new product may penetrate the consumer market. We review the traditional approach in the marketing literature, which is based on differential or difference equations similar to the logistic equation (Bass 1969). This mean field approach is contrasted with the discrete percolation on a lattice, with simulations of "social percolation" (Solomon et al 2000) in two to five dimensions giving power laws instead of exponential growth, and strong fluctuations right at the percolation threshold.Comment: to appear in Physica

    3D view of transient horizontal magnetic fields in the photosphere

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    We infer the 3D magnetic structure of a transient horizontal magnetic field (THMF) during its evolution through the photosphere using SIRGAUS inversion code. The SIRGAUS code is a modified version of SIR (Stokes Inversion based on Response function), and allows for retrieval of information on the magnetic and thermodynamic parameters of the flux tube embedded in the atmosphere from the observed Stokes profiles. Spectro-polarimetric observations of the quiet Sun at the disk center were performed with the Solar Optical Telescope (SOT) on board Hinode with Fe I 630.2 nm lines. Using repetitive scans with a cadence of 130 s, we first detect the horizontal field that appears inside a granule, near its edge. On the second scan, vertical fields with positive and negative polarities appear at both ends of the horizontal field. Then, the horizontal field disappears leaving the bipolar vertical magnetic fields. The results from the inversion of the Stokes spectra clearly point to the existence of a flux tube with magnetic field strength of 400\sim400 G rising through the line forming layer of the Fe I 630.2 nm lines. The flux tube is located at around logτ5000\log\tau_{500} \sim0 at Δt\Delta t=0 s and around logτ5001.7\log\tau_{500} \sim-1.7 at Δt\Delta t=130 s. At Δt\Delta t=260 s the horizontal part is already above the line forming region of the analyzed lines. The observed Doppler velocity is maximally 3 km s1^{-1}, consistent with the upward motion of the structure as retrieved from the SIRGAUS code. The vertical size of the tube is smaller than the thickness of the line forming layer. The THMF has a clear Ω\Omega-shaped-loop structure with the apex located near the edge of a granular cell. The magnetic flux carried by this THMF is estimated to be 3.1×10173.1\times10^{17} Mx.Comment: 35 pages, 9 figures, Accepted for publication in Ap

    Longitudinal flow evolution and turbulence structure of dynamically similar, sustained, saline density and turbidity currents

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    Experimental results are presented concerning flow evolution and turbulence structure of sustained saline and turbidity flows generated on 0°, 3°, 6°, and 9° sloping ramps that terminate abruptly onto a horizontal floor. Two-component velocity and current density were measured with an ultrasonic Doppler velocity profiler and siphon sampler on the slope, just beyond the slope break and downstream on the horizontal floor. Three main factors influence longitudinal flow evolution and turbulence structure: sediment transport and sedimentation, slope angle, and the presence of a slope break. These controls interact differently depending on flow type. Sediment transport is accompanied by an inertial fluid reaction that enhances Reynolds stresses in turbidity flows. Thus turbidity flows mix more vigorously than equivalent saline density flows. For saline flows, turbulent kinetic energy is dependent on slope, and rapid deceleration occurs on the horizontal floor. For turbidity flows, normalized turbulent kinetic energy increases downstream, and mean streamwise deceleration is reduced compared with saline flows. The slope break causes mean bed-normal velocity of turbidity flows to become negative and have a gentler gradient compared with other locations. A reduction of peak Reynolds normal stress in the bed-normal direction is accompanied by an increase in turbulent accelerations across the rest of the flow thickness. Thus the presence of particles acts to increase Reynolds normal stresses independently of gradients of mean velocity, and sediment transport increases across the break in slope. The experiments illustrate that saline density currents may not be good dynamic analogues for natural turbidity currents

    Two step ejection of massive stars and the issue of their formation in isolation

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    In this paper we investigate the combined effect of massive binary ejection from star clusters and a second acceleration of a massive star during a subsequent supernova explosion. We call this the "two-step-ejection" scenario. The main results are: i) Massive field stars produced via the two-step-ejection process can not in the vast majority of cases be traced back to their parent star clusters. These stars can be mistakenly considered as massive stars formed in isolation. ii) The expected O star fraction produced via the two-step-ejection process is of the order of 1-4 per cent, in quantitative agreement with the observed fraction of candidates for isolated-O-star formation. iii) Stars ejected via the two-step-ejection process can get a higher final velocity (up to 1.5-2 times higher) than the pre-supernova velocity of the massive-star binary.Comment: Accepted by MNRA

    Role of the Fractalkine Receptor in CNS Autoimmune Inflammation: New Approach Utilizing a Mouse Model Expressing the Human CX3CR1

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    Multiple sclerosis (MS), an inflammatory demyelinating disease of the central nervous system (CNS) is the leading cause of non-traumatic neurological disability in young adults. Immune mediated destruction of myelin and oligodendrocytes is considered the primary pathology of MS, but progressive axonal loss is the major cause of neurological disability. In an effort to understand microglia function during CNS inflammation, our laboratory focuses on the fractalkine/CX3CR1 signaling as a regulator of microglia neurotoxicity in various models of neurodegeneration. Fractalkine (FKN) is a transmembrane chemokine expressed in the CNS by neurons and signals through its unique receptor CX3CR1 present in microglia. During experimental autoimmune encephalomyelitis (EAE), CX3CR1 deficiency confers exacerbated disease defined by severe inflammation and neuronal loss. The CX3CR1 human polymorphism I249/M280 present in ∼20% of the population exhibits reduced adhesion for FKN conferring defective signaling whose role in microglia function and influence on neurons during MS remains unsolved. The aim of this study is to assess the effect of weaker signaling through hCX3CR1I249/M280 during EAE. We hypothesize that dysregulated microglial responses due to impaired CX3CR1 signaling enhance neuronal/axonal damage. We generated an animal model replacing the mouse CX3CR1 locus for the hCX3CR1I249/M280 variant. Upon EAE induction, these mice exhibited exacerbated EAE correlating with severe inflammation and neuronal loss. We also observed that mice with aberrant CX3CR1 signaling are unable to produce FKN and ciliary neurotrophic factor during EAE in contrast to wild type mice. Our results provide validation of defective function of the hCX3CR1I249/M280 variant and the foundation to broaden the understanding of microglia dysfunction during neuroinflammation. © 2018 Cardona et al
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